Neonatal Resuscitation: Nrp Slide Presentation Kit

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It is appropriate to feed infants who have hyperglycemia unless other clinical problems are considered severe enough to prevent feeding. The plasma glucose concentration relative to the rate of IV glucose administered in a representative neonate in the 500-g birthweight category (a) and in one in the 900-g birthweight category (b). If severe hyperglycemia persists, exogenous insulin administration may be warranted. From author Brian Walsh, an experienced educator and respiratory therapist, this text is an excellent study tool for the NBRC's Neonatal/Pediatric Specialty exam!

Pages: 0

Publisher: Amer Academy of Pediatrics; 5th edition (May 2006)

ISBN: 1581102003

An IV is started and 8 ml (2 mg/kg) of 10% dextrose in water (D10W) is given IV. The infant is transferred to the intermediate nursery where a repeat blood sugar 30 minutes after the bolus is still <20 mg/dl. The earlier serum glucose sent to the lab, comes back at <2 mg/dl. IV D10 is infusing at 80 ml/kg/day, which is a glucose infusion rate of 5.6 mg/kg/min , cited: read online. In clinical circumstances, the definition typically is based upon the hct, rather than the hemoglobin concentration. The diagnosis of polycythemia is based upon peripheral venous samples because of the variability in measurements obtained from capillary samples. Hcts of blood from venous samples may be as much as 15 percent lower than those obtained from capillary samples. (See 'Laboratory testing' below.) Hyperviscosity — Polycythemia must be distinguished from hyperviscosity, which is defined as a blood viscosity >12 centipoise, measured at a shear rate of 11.5 per second; or >6 centipoise, measured at a shear rate of 106 per second [ 7 ] hudsoncriminaldefenselawyer.com. Post-hypoxic hypothermia reduces cerebrocortical release of NO and excitotoxins. Neuroreport. 1997;8:3359-3362 PMID:9351672 12. Specific inhibition of apoptosis after cerebral hypoxia-ischaemia by moderate post-insult hypothermia. Biochem Biophys Res Commun. 1995;217:1193-1199. Apoptosis in perinatal hypoxic-ischemic brain injury: how important is it and should it be inhibited , e.g. http://postdialysisfatigue.net/lib/understanding-craniofacial-anomalies-the-etiopathogenesis-of-craniosynostoses-and-facial-clefting?
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